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		<title>脑梗死-发病机制-脑组织出现严重的缺血坏死 - 版本历史</title>
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		<updated>2026-04-22T02:31:16Z</updated>
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		<title>Admin：修改1</title>
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				<updated>2024-07-06T14:00:00Z</updated>
		
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		<author><name>Admin</name></author>	</entry>

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		<title>KGadmin：导入1个版本</title>
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		<summary type="html">&lt;p&gt;导入1个版本&lt;/p&gt;
&lt;p&gt;&lt;b&gt;新页面&lt;/b&gt;&lt;/p&gt;&lt;div&gt;主实体：[[脑梗死]]，关系：[[发病机制]] ，尾实体：[[脑组织出现严重的缺血坏死]]&lt;br /&gt;
&lt;br /&gt;
实体ID:''10917088''&lt;br /&gt;
==关系介绍==&lt;br /&gt;
待补充&lt;br /&gt;
&lt;br /&gt;
==来源==&lt;br /&gt;
===辛伐他汀对急性脑梗死患者血清CRP及IL-6水平的影响探讨（中华医学会文献）===&lt;br /&gt;
张建磊等[18]研究报道中表明，患者出现[[脑梗死]]后，因为[[脑组织出现严重的缺血坏死]]，激活了很多抗原免疫系统，而且也激活了很多炎症细胞，大大增加了单核巨噬细胞、B淋巴细胞、T淋巴细胞等活性，导致大量IL-6产生，进而使血管通透性降低，使血细胞黏附性以及血管内皮细胞黏附性增加，提高人体中自由基含量，进而促进细胞死亡。&lt;/div&gt;</summary>
		<author><name>KGadmin</name></author>	</entry>

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